Liver hypertrophy in children
This disease affects adults and young people, the liver becomes larger than normal size and today we will talk about this disease in children.
Liver hypertrophy is usually a sign of a latent problem. There are many possible causes of liver inflation, including hepatitis, depending on the cause of hepatic hyperplasia sometimes leading to liver failure, and this occurs when a large part of the liver is damaged and deteriorates and falls on The right side of the abdomen, the liver is a vital organ in the human body For example, it helps to rid the body of toxins and produce substances that help in the body’s vital processes such as coagulation
Causes of liver enlargement:
– fatty liver inflammation
– The presence of infection such as virus or abscess
– some medicine
– Toxins
– Certain types of hepatitis including alcohol and hepatitis
– autoimmune diseases
– Metabolic syndrome
– Genetic disorders that cause fat, protein, or other substances to build
– abnormal growth may cause liver enlargement
– Cysts
– Tumors that begin or spread to the liver
Congestive heart failure, a condition in which the heart fails to pump blood well
– Hepatic venous thrombosis
– blockage of veins in the liver
– An obstructive vein disease, clogging of small veins in the liver
Symptoms of hepatic hyperplasia:
In most cases, hepatic hyperplasia does not cause any symptoms, however, it appears:
– Feeling full
– Abdominal pain
– Jaundice
– Fatigue
– Weakness
– Nausea
– Weight loss
– During physical examination, your doctor can detect liver hypertrophy in the right side of the abdomen
A variety of tests will also be needed to confirm the cause of liver hyperplasia, including:
– Blood tests to confirm liver enzymes and other abnormalities related to liver diseases
– CT scan and magnetic resonance imaging (MRI), or ultrasound display
The liver is a range test to check for bile duct problems which is a MRI test for a special examination within the bile duct biopsy
– Examine a sample of liver tissue to check for cancer or fatty liver
Treatment of liver hyperplasia:
Without treatment, liver hypertrophy can lead to liver failure Treatment for liver hyperglycemia depends on the cause For example, if drinking too much alcohol is the source of the problem, it must be stopped so that the liver does not turn severely damaged.
Prevention of liver enlargement:
– In order to reduce the risk of liver disease, you can:
– Choose a healthy diet. Choose a healthy diet rich in fruits, vegetables and whole grains.
– Avoid drinking alcohol
Follow instructions and instructions when taking drugs, vitamins, or supplements. Do not use any overdose recommended when taking vitamins, supplements or prescription or non-prescription medicines.
– Reduce the handling of chemicals. Aerosol cleaners, insecticides and toxic chemicals should only be used in well ventilated areas. In addition, wear gloves, long sleeves and face masks.
– Maintain healthy weight. If you have the ideal weight and health, you have to keep it. If you need to lose weight / reduce the number of calories you eat daily, exercise more daily. Ask your doctor about healthy ways to help you lose weight.
– To stop exaggeration. If you smoke, you should immediately stop smoking. Ask your doctor about some strategies that you can use to stop smoking. If you do not smoke, you never think about it.
– Use the supplements very carefully. Talk to your doctor about the risks and benefits of herbal supplements before taking them. Some alternative medical treatments may be detrimental to liver health, including black cohosh, some Chinese herbs that include mahwing, grapefruit, senfite, mistletoe, crumple, keratin, kava, catnip,
Studies and research on liver hypertrophy:
– Eating high-carbohydrate-rich foods has been linked to the disease and eating too high a diet rich in potatoes, white bread and white rice may contribute to hepatic liver disease, according to a study by researchers at the Boston Children’s Hospital and published in the journal Obesity. Fatty) as a result of the accumulation of fat around it which is similar to the silent disease, which affects serious complications.
Those who were fed the same amount of other foods did not catch the disease
An obesity expert said the liver fat in children would be “the tragedy of the future.”
This may not have a satisfactory effect on the patient at this stage, but this condition is associated with a greater risk of injury to the owner of liver failure later in life.
The study looked at the effect of calorie-content diets, but with different ingredients when measured using the “Glucose Index” (GI), a measure of the speed of energy absorption in food by the body, which increases blood sugar levels.
Foods high in sugars cause a sharp rise in blood sugar levels and a similar rise in insulin levels produced by the pancreas
High foods on the sugary index include many cornflakes and processed foods, such as white bread and white rice.
While low-sugar foods include unprocessed fruits, nuts, grains and spaghetti.
Dr David Ludwig, who led the research, said he believed that between a quarter and a half of obese children in the United States suffer from liver disease.
“This is a silent but serious disease,” he said.
“Just as the second type of diabetes suddenly emerged in the 1990s, we also believe that liver disease will have the same effect over the next decade,” he said.
The biggest threat to foods rich in sugars is insulin resistance – the first signs of type 2 diabetes, said Azmina Govindja, a nutritionist and spokeswoman for the British Dietetic Association.
– A study showed that all acute liver patients suffer from fatigue, abdominal pain, erythema, fever, loss of appetite, liver enlargement and spleen enlargement in 70%, while most chronic liver patients had all the symptoms in addition to the enlargement of the spleen at different rates .. The loss Weight, ascites and bleeding have been observed in patients with chronic liver disease.
Blood factor: The lack of hemoglobin, number of red balls and blood deposits in all patients were found to reflect anemia. In acute liver patients, the mean mean was 1, 8 ± 4, 2 g / dL, 9, 2 ± 9, 0 X 1210 / L, 9, 27 ± 7 and 9%. In chronic liver patients, it was 4, 9 ± 0, 2 g / dec, 5, 3 ± 8, 5 X 1210 / For 4, 30 ± 9 and 6%. The quality of anemia was anemia with red cells of normal size and redness, indicating an anemia caused by chronic disorders. It was noted that the incidence of anemia in chronic liver patients (22 and 51%) was especially in patients with hepatocellular carcinoma (80% 10%). Small red fever anemia was observed in patients with chronic liver disease (83% and 26%) in general and in cirrhosis patients (6 and 34%) and acute liver disease (20%).
(60%) and chronic hepatocellular disease (51% and 19%), which reflect the presence of hydrolysis in these patients.
The incidence of anemia varies from simple to severe, with most of the patients with chronic liver disease (4 and 86%). In acute liver patients, the percentage was 6% and 13%. Average anemia was observed in chronic liver patients (2 and 76% And acute liver disease (8% and 23%).
Acute anemia was found in patients with chronic liver disease (7% and 66%) and in acute liver patients (3 and 33%).
White blood cells were normal, as they were found in patients with acute liver 3, 7 ± 8, 5 X 610 / L and in chronic liver patients 9, 6 ± 7 and 4 X 610 / l. White blood cell deficiency was observed in 50% of acute hepatic patients and 60% of cirrhosis patients with spleen. As for cell augmentation, 30% of patients with hydrolysis and bacterial infections were found. The standard mean of platelets was normal. In acute liver patients 9, 153 ± 7 and 66 X 910 / L and in chronic liver patients 0, 150 ± 2 and 89 X 910 / l. Platelet deficiency was found in patients with cirrhosis. Retinal red cells were found to be high in patients with acute liver 5, 2 ± 8 and 1%.
The mean mean red blood cell density was high in acute liver patients 2, 67 ± 0, 44 mm / o and chronic liver disease 7, 57 ± 8 and 39 mm / o, especially in patients with hepatocellular carcinoma. The infected red cell was found only in patients with cirrhosis. The target cell was found more in acute liver patients.
There were statistically significant differences in the number of red blood cells and the number of retinal cells among acute and chronic liver patients. The number of platelets, white blood cells and sedimentation rate was among chronic liver patients.